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Work-related asthma is a serious condition indeed. Asthma
affects your life and well-being more than many other
afflictions. It is a condition which may be very difficult to
live with and impossible to come to terms with.
In serious cases, asthma is a killer disease and every year
people die from it.
What is asthma? In short, it may be explained as a reduction
in the diameter of the bronchi. By muscular contraction, this
reduction hinders the passage of air, and sometimes closes it off
entirely.
The bronchial "tightening" is caused by various
agents that are triggered by allergic reactions, physical
irritation, neurogenic phenomena, or pharmacodynamic agents.
In addition to the blocking of the bronchi, there is also
hypersensitivity and inflammation of the mucous membranes. The
hypersensitivity can render the asthmatic person extremely
susceptible to the particular substance and even minute amounts
may cause a severe attack.
Occupational asthma is often connected with other allergic
reactions, such as hay fever, rhinitis, coughs, etc What makes it
a work-related illness is that it is brought about by single or
repeated exposure to substances present in the working
environment.
SYMPTOMS
An asthma attack often starts after work, sometimes in the
evening, often at night, most commonly in the early hours of the
morning. The asthmatic usually wakes up with an initial sense of
anxiety even panic
coupled with pressure about the chest, coughing and a
runny nose. The impaired breathing is further compounded by
phlegm and secretion. Acute asthma attacks may require urgent
resuscitation and emergency medical assistance.
Treatment is difficult. Antihistamine and other preparations
(such as the common inhalation spray tubes) may be used to milden
the attack and relieve the breathing impairment
but these are only used for symptomatic treatment, In
order to treat the asthma itself, immunological desensitising
treatment is necessary. However, this may prove very difficult
and, in some situations, downright dangerous. Many people are
left to live with their condition for the rest of their lives.
PREVENTION
The main effective means of prevention is to examine which
airborne particles are present in the workplace and then to
determine whether these particles are capable of causing asthma.
If so, radical steps should be taken to a) replace the substance
with a non-allergenic one b) engineer out the problem through
mechanical exhaust, improved ventilation, etc., and c) determine
the type of respirator needed, and make sure that all workers
wear one.
WHEN IS ASTHMA OCCUPATIONAL?
This question is often a "hot potato", because it is
not always possible to clearly define the cause of the illness.
The presence of the allergen may not be clear-cut, for instance,
if the asthma is triggered by a substance that occurs in high
concentrations at work, but also is generally dispersed
elsewhere.
To our knowledge conclusive guidelines have yet to be
determined. However, here is a short list of normally accepted
criteria as to what is and what
isnt occupational asthma:
ASTHMA IS OCCUPATIONAL:
- When there is only one trigger substance and that
substance occurs at work, and the asthma disappears when
work is stopped.
- When an irritant substance causes the asthma, and the
asthma disappears when the occupation is terminated or
changed.
- When the person is sensitive to a substance that occurs
both at work and at home but does not experience
asthmatic attacks after finishing work. In this case the
asthma is still regarded as occupational, because the
work environment creates the additional exposure that
results in an attack.
- When the person is sensitive to a substance not present
at work, but whose attacks are created by an irritant
found at work. This is still treated as occupational
asthma, because the patient recovers after leaving work.
...BUT NOT OCCUPATIONAL:
The list of asthma-producing substances is already very long,
and growing rapidly. Here are just a few well-known agents of
occupational asthma:
- Acrylic fibre
- Alkyl phosphates
- Aliphatic aldehydes
- Aliphatic amines
- Ampicillin
- Azo dyes
- Barley
- Budgerigars
- Carbamates
- Castor Oil
- Cats
- Chlorine
- Chlorthion
- Chromium
- Cobalt
- Colophony fumes
- Cotton
- Cows
- Diazinon
- Diazomethane
- Diethanolamine
- Diethylene diamine
- Diethylenetriamine
- Dogs
- Epoxy resins & hardeners
- Ethyl hexylamine
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- Flour
- Formaldehyde
- Grain
- Ground nuts
- Gum arabic
- Hair, feathers
- Hemp
- Henna
- Insecticides
- Jute
- Lead
- Liquorice
- Mercury diphenyl
- Mercury, organic compounds
- Metampicillin
- Mites
- Mould
- Oats
- Organic isocyanates
- Organiphosphorous compounds
- p-Dichlorobenzene
- p-Formaldehyde
- Penicillin
- Persulphates
- Pesticides
- p-Phenyldiamine
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- Phenyl-formaldehyde resins
- Phenylglycine
- Phenylhydrazine
- Phosphoramines
- Phthalic acid
- Piperazine
- Platinum salts
- Polyamides
- Polyesters
- Proteolytic enzymes
- Pyrethrum
- Quinine
- Rice
- Rye
- Sericin
- Silk
- Soya
- Textiles
- Toluenediisocyanate
- Triethylenediamine
- Triethylenetetramine
- Trypsin
- Urea-formaldehyde resins
- Vanadium
- Vanillin
- Welding fumes
- Wood
- Wool
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Sources: Encyclopaedia of Occupational Health
and Safety, International Labour Office, Geneva 1985, Black's
Medical Dictionary, 35th ed., London 1987; Chemical Hazards at
the Workplace, Proctor and Hughes, Philadelphia 1978
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